Author

Shannon E. Niedermeyer

Johns Hopkins UniversityORCID 0000-0003-1804-7125Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Bioinformatics· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:benchmarking· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Approach:machine learning (classical)· 1

Frequent Co-Authors

Karthik S. SureshiD· 2 shared Larissa A. ShimodaiD· 2 shared Michael P. CroglioiD· 2 shared Catherine E. SimpsoniD· 1 shared Christopher CherryiD· 1 shared Darin T. RoseniD· 1 shared Haiyang JiangiD· 1 shared John HuetschiD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersPhysiology

Karthik S. SureshORCID

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2 papers

Recent·Most cited

PhysiologybioRxivPreprintNov 6, 2025

Na+/H+ Exchanger Isoform 1 Regulates Apoptosis Susceptibility in Pulmonary Arterial Smooth Muscle from the Sugen/Hypoxia model of Pulmonary Hypertension

Manuella Ribas Andrade, Xin Yun, Michael P. Croglio…·Larissa A. Shimoda

This study shows NHE1 promotes apoptosis resistance in PASMCs from PH models through p38 suppression, independent of ion transport or ezrin-binding.

DOI 10.1101/2025.11.06.687041

ratexperimental assay

𝕏 PostBluesky

BioinformaticsbioRxivPreprintOct 25, 2024

Single-cell transcriptomics reveal diverging pathobiology and opportunities for precision targeting in scleroderma-associated versus idiopathic pulmonary arterial hypertension

Tijana Tuhy, Julie C. Coursen, Tammy Graves…·Catherine E. Simpson

This study uses single-cell RNA sequencing to compare scleroderma-associated and idiopathic pulmonary arterial hypertension, identifying distinct pathobiological features and potential drug targets.

2 citations

𝕏 PostBluesky