Author

Timothy R. Matsuura

University of PennsylvaniaORCID 0000-0003-1813-6451Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Physiology· 2 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:drug response / drug discovery· 2 Assay:metabolomics· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Organism:human· 1

Frequent Co-Authors

Daniel P. KellyiD· 3 shared Jiten D. PateliD· 2 shared Justin H. BergeriD· 2 shared Renee Taing· 2 shared Attila KovácsiD· 1 shared Bei B. Zhang· 1 shared Caitlyn E. BowmaniD· 1 shared Dinesh K. Hirenallur-ShanthappaiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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3 shared papersPhysiology

Daniel P. KellyORCID

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3 papers

Recent·Most cited

NeurosciencebioRxivPreprintMar 27, 2026

Requirement for oxidation of neuronal ketone bodies in aging andneurodegeneration

Joyce L. Yang, Mitsunori Nomura, Jonathan Xianglong Meng…·John C. Newman

This study shows ketone body oxidation is essential for neuronal function and survival, particularly in neurodegenerative diseases like Alzheimer's.

1 citation

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PhysiologyScientific ReportsPeer-reviewedJan 2, 2025

Two-hit mouse model of heart failure with preserved ejection fraction combining diet-induced obesity and renin-mediated hypertension

Justin H. Berger, Yuji Shi, Timothy R. Matsuura…·Daniel P. Kelly

This paper develops a two-hit mouse model of HFpEF combining diet-induced obesity and renin-mediated hypertension, showing key features of the disease and response to therapy.

21 citationstop 1%

mousebulk RNA-seq

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PhysiologyCirculation ResearchPeer-reviewedJul 23, 2024

Sodium-glucose co-transporter 2 Inhibitors Act Independently of SGLT2 to Confer Benefit for Heart Failure with Reduced Ejection Fraction in Mice

Justin H. Berger, Timothy R. Matsuura, Caitlyn E. Bowman…·Daniel P. Kelly

This study shows that SGLT2 inhibitors improve heart failure in mice independently of their metabolic effects, even when SGLT2 is absent.

47 citationstop 1%

mousemetabolomics

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