Holger Haselmann

This study shows that autoantibodies against GABAB receptors cause presynaptic pathology leading to cognitive decline and memory impairment.

This study shows that disrupted migration of inhibitory neurons leads to persistent functional deficits in cortical networks, linking migratory guidance to circuit integrity.

This study investigates how anti-NMDAR encephalitis alters neuronal network function by enhancing coupling and clustering in hippocampal neurons during slow-wave sleep.

This study shows that NMDAR autoantibodies disrupt the excitatory-inhibitory balance in the hippocampus, leading to network hypersynchrony and symptoms resembling schizophrenia.

This study identifies synaptic pathomechanisms of sepsis-associated encephalopathy and demonstrates that targeted rescue of synaptic plasticity can improve cognitive decline in a mouse model.