Selective depletion of AMH-expressing granulosa cells in vivo impairs follicular development and fertility in female mice
Tsutomu Endo, Mayu Tamemasa, Kana Hayakawa…·Masami Kanai‐Azuma
Author
The University of TokyoORCID 0000-0003-2116-7806Unclaimed ORCID profile · follow to get notified of new preprints
Mutual co-authors in cluster
6 papers
Recent·Most cited
Tsutomu Endo, Mayu Tamemasa, Kana Hayakawa…·Masami Kanai‐Azuma
Han Xiao, Aya Uchida, Seohyeon Lee…·Yoshiakira Kanai
This study shows that ectopic SOX17 in Sertoli cells promotes Sertoli valve formation and partially rescues infertility in Sox17-null mice.
Yuta Uneme, Ryu Maeda, Gen Nakayama…·Soichiro Yamanaka
The paper shows Morc1 re-silences activated transposons in gonocytes by establishing heterochromatin, preventing their disruptive effects on the transcriptome.
Gerel Melig, Ikuo Nobuhisa, Kiyoka Saito…·Tetsuya Taga
This study identifies Rasip1 as a downstream target of Sox17 that enhances hematopoietic activity in intra-aortic hematopoietic clusters of midgestation mouse embryos.
Rie Saba, Keiko Kitajima, Lucille Rainbow…·Kenta Yashiro
This study shows that Sox17 expression in cardiac progenitor cells biases their fate toward the endocardium and regulates myocardium development through NOTCH signaling in mice.
Risa Nomura, Kenichi Kashimada, Hitomi Suzuki…·Tomohiro Morio
This paper shows that Nr5a1 suppression during fetal development optimizes ovarian development by fine-tuning Notch signaling, using mouse models with altered Nr5a1 expression.