Erin E. Young

The paper identifies Avpr1a as a novel candidate gene for chronic visceral pain through genomic and functional analyses in mouse models and human patients.

This study shows that spinal cord injury rapidly alters nociceptor response properties, leading to increased sensitivity and spontaneous activity, contributing to chronic pain.

This study shows TIMP-1 reduces inflammatory pain through both MMP-dependent and receptor-mediated signaling mechanisms.

This study identifies a Trpa1 splice junction variant in Diversity Outbred mice that influences late phase formalin response, linking it to pain sensitivity mechanisms.

The paper uses single cell qPCR and functional assays to correlate gene expression with functional properties in identified sensory neurons, finding transcriptional heterogeneity within functional subgroups.