Author

Kinulpe Honorato Sampaio

Universidade Federal dos Vales do Jequitinhonha e MucuriORCID 0000-0003-2622-032XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

B. d. L. SanchesiD· 1 shared Diogo B. PeruchettiiD· 1 shared Fábio César Sousa NogueiraiD· 1 shared Itamar Couto Guedes de JesusiD· 1 shared Jáder Santos CruziD· 1 shared Joseph Albert Medeiros EvaristoiD· 1 shared Júlia Rezende-Ribeiro· 1 shared Lucas Rodrigues‐RibeiroiD· 1 shared

Activity by Year

2024-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPhysiology

B. d. L. SanchesORCID

Mutual co-authors in cluster

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1 paper

Recent·Most cited

Evidence that the monoamine oxidase B (MAO-B) plays a central role in the inotropic dysfunction induced by genetic deletion of the Mas-related-G protein-coupled receptor D (MrgD) in mice

Lucas Rodrigues‐Ribeiro, Júlia Rezende-Ribeiro, Sérgio Scalzo, Maria Luiza Dias, B. d. L. Sanches, Marcos Eliezeck, Itamar Couto Guedes de Jesus, Joseph Albert Medeiros Evaristo, Kinulpe Honorato Sampaio, Diogo B. Peruchetti, Jáder Santos Cruz, Fábio César Sousa Nogueira, Maria José Campagnole‐Santos, Sílvia Guatimosim, Robson A.S. Santos, Thiago Verano‐Braga

This study identifies MAO-B hyperactivation as a key contributor to cardiac dysfunction in MrgD-KO mice, showing that MAO-B inhibition reverses contractile defects.

bioRxiv · Mar 27, 2024 · PreprintPhysiology