Author

Qasim A. Majid

Imperial College LondonORCID 0000-0003-2720-9149Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 1 Neuroscience· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell-cell communication· 2 Assay:bulk RNA-seq· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1

Frequent Co-Authors

Heikki J RuskoahoiD· 2 shared Katriina Aalto‐SetäläiD· 2 shared Virpi TalmaniD· 2 shared Arnau HerveraiD· 1 shared Chandra Prajapati· 1 shared Francesco De VirgiliisiD· 1 shared Helen LeonardiD· 1 shared Ilaria PalmisanoiD· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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Focal author (center)Selected co-authorORCID co-authorUnlinked name

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2 shared papersPharmacology & Toxicology

Heikki J RuskoahoORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Pharmacology & ToxicologybioRxivPreprintSep 4, 2026

Patient-Derived hiPSC-Cardiomyocytes and Engineered Heart Tissues Reveal Distinct Functional Phenotypes in Inherited Cardiomyopathies

Saana Pohjavaara, Qasim A. Majid, Leo Huttunen…·Virpi Talman

This study uses patient-derived hiPSC-cardiomyocytes and engineered heart tissues to compare functional and molecular phenotypes in HCM and DCM, revealing genotype-specific responses to stimuli.

DOI 10.64898/2026.08.28.747951

humanscRNA-seq

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Molecular BiologybioRxivPreprintFeb 19, 2025

The novel SCN5A-P1891A mutation is associated with left ventricular hypertrabeculation and links Nav1.5 to cardiomyocyte proliferation and disrupted 3D cardiac tissue formation

Qasim A. Majid, Chandra Prajapati, Xiaonan Liu…·Virpi Talman

This study identifies a novel SCN5A mutation linked to left ventricular hypertrabeculation and shows its impact on cardiomyocyte proliferation and 3D cardiac tissue formation.

DOI 10.1101/2025.02.19.639199

humanbulk RNA-seq

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NeurosciencebioRxivPreprintJul 2, 2019

Cyclin-dependent-like kinase 5 is required for pain signalling in both human neurons and mouse models

Paolo La Montanara, Arnau Hervera, Lucas L. Baltussen…·Simone Di Giovanni

This study identifies Cdkl5's role in pain signaling through its interaction with CaMKII in both human neurons and mouse models of Cdkl5 deficiency disorder.

DOI 10.1101/690172

humanmouse

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