Author

Michaël Pollak

Jewish General HospitalORCID 0000-0003-3047-0604Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cancer Biology· 2 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 3 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Research Focus:GWAS / association mapping· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Anastasija Ana Piric· 1 shared Andia Moshari· 1 shared Binadi Diddeniya· 1 shared Branka VuleseviciD· 1 shared Celia M.T. GreenwoodiD· 1 shared Charlotte GuzmaniD· 1 shared Chris Tselios· 1 shared Clare PatersoniD· 1 shared

Activity by Year

2021-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

Anastasija Ana Piric

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cancer BiologybioRxivPreprintSep 16, 2026

A therapeutic vulnerability linking MNK1/2 inhibition and G1/S cyclin-dependent kinase blockade in triple-negative breast cancer

Qiyun Deng, Mehdi Amiri, Andia Moshari…·Nahum Sonenberg

This study identifies a synergistic therapeutic effect of combining MNK1/2 inhibition with G1/S CDK blockade in triple-negative breast cancer.

DOI 10.64898/2026.09.11.750258

humanbulk RNA-seq

𝕏 PostBluesky

Cancer BiologyeLifePeer-reviewedMay 5, 2026

Mitochondrial ETF insufficiency drives neoplastic growth by selectively optimizing cancer bioenergetics

David J. Papadopoli, Ranveer Palia, Predrag Jovanovic…·Ivan Topisirović

This study shows that mitochondrial ETF insufficiency promotes cancer growth by reprogramming bioenergetics and signaling pathways in neoplastic cells.

1 citation

humanexperimental assay

𝕏 PostBluesky

Genetic & Genomic MedicineNature MedicinePeer-reviewedFeb 25, 2021

A Neanderthal OAS1 Isoform Protects Against COVID-19 Susceptibility and Severity: Results from Mendelian Randomization and Case-Control Studies

Sirui Zhou, Guillaume Butler‐Laporte, Tomoko Nakanishi…·John Brent Richards

This study identifies OAS1 as a protective factor against COVID-19 severity and susceptibility using Mendelian randomization and case-control analyses.

273 citationstop 1%

humanproteomics

𝕏 PostBluesky