Author

David J. Lamb

Boehringer Ingelheim (Germany)ORCID 0000-0003-3097-6189Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Anissa Anindya WidjajaiD· 1 shared Benjamin L. GeorgeiD· 1 shared Brijesh Kumar SinghiD· 1 shared David CarlingiD· 1 shared Dong Jinrui· 1 shared Eleonora AdamiiD· 1 shared Jessie Tan· 1 shared Joyce Wei Ting GohiD· 1 shared

Activity by Year

2021-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperMolecular Biology

Anissa Anindya WidjajaORCID

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1 paper

Recent·Most cited

Molecular BiologybioRxivPreprintJun 10, 2021

Molecular dissection of pro-fibrotic signaling identifies the mechanism underlying IL11-driven fibrosis gene translation, reveals non-specific effects of STAT3 and suggests a new mechanism of action for nintedanib

Anissa Anindya Widjaja, Sivakumar Viswanathan, Dong Jinrui…·Stuart A. Cook

The paper investigates IL11-driven fibrosis mechanisms, showing ERK, not STAT3, drives fibrogenesis and identifies a novel role for nintedanib in inhibiting pro-fibrotic translation.

DOI 10.1101/2021.06.10.447846

mouseproteomics

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