Author

Yegor S Vassetzky

Centre National de la Recherche ScientifiqueORCID 0000-0003-3101-7043Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Anna A. KarpukhinaiD· 2 shared Antonin MarchaisiD· 1 shared Azat K GaraeviD· 1 shared Birgit GeoergeriD· 1 shared Catherine BrenneriD· 1 shared Daniela Dias‐PedrosoiD· 1 shared Daria Bogdanova· 1 shared Diego GerminiiD· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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2 shared papersCancer Biology

Anna A. KarpukhinaORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Cancer BiologyCancer LettersPeer-reviewedJul 17, 2026

RNAseq analysis reveals the recurrent loss of heterozygosity in lung cancer and associated transcription patterns

Ruslan Gumerov, Wangzhen He, Phong Luong…·Anna A. Karpukhina

This study uses RNA-seq to infer chromosomal aberrations in lung cancer and identifies shared and deletion-specific transcriptional patterns and biomarkers.

DOI 10.1016/j.canlet.2026.218741

humanbulk RNA-seq

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Cancer BiologybioRxivPreprintJun 9, 2026

Translocation-driven chr19-der(14) interaction is associated with disease-specific transcriptional programs in mantle cell lymphoma

Anna A. Karpukhina, Yelena Grishina, Ivan Tsimailo…·Yegor S Vassetzky

This study identifies interchromosomal interactions between chr19 and the CCND1 locus in MCL, linking spatial genome reorganization to disease-specific transcriptional programs.

DOI 10.64898/2026.06.05.730311

humanbulk RNA-seq

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Cancer BiologybioRxivPreprintApr 7, 2026

Combined inhibition of AIF/CHCHD4 interaction and GLS1 to exploit metabolic vulnerabilities in pediatric osteosarcoma

Hong-Toan Lai, Tran Ngoc Anh Nguyen, Maria Eugénia Marques Da Costa…·Catherine Brenner

This study identifies a metabolic vulnerability in osteosarcoma by combining AIF/CHCHD4 inhibition and GLS1 suppression, showing synergy between mitoxantrone and telaglenastat in vitro and in vivo.

DOI 10.64898/2026.04.03.716303

humanmetabolomics

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