Author

Michael C. Gundry

Baylor College of MedicineORCID 0000-0003-3654-1741Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:gene regulatory network inference· 1 Research Focus:variant calling· 1 Research Focus:variant effect prediction· 1

Frequent Co-Authors

Andrea ArrudaiD· 1 shared Chenghang Chuck ZongiD· 1 shared Claudia A. MimosoiD· 1 shared Fiona E. ReediD· 1 shared Jennifer A. PerryiD· 1 shared João A. PauloiD· 1 shared Karen AdelmaniD· 1 shared Kellie R. MachlusiD· 1 shared

Activity by Year

2019-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperCancer Biology

Andrea ArrudaORCID

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2 papers

Recent·Most cited

Cancer BiologyBloodPeer-reviewedSep 24, 2025

CEBPA repression by MECOM blocks differentiation to drive aggressive leukemias

Travis J. Fleming, Mateusz Antoszewski, Sander Lambo…·Vijay G. Sankaran

This study identifies MECOM's role in blocking differentiation via CEBPA repression, showing that targeting this regulatory element can promote differentiation and reduce leukemia burden in AMLs.

11 citationstop 2%

humanbulk RNA-seq

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GenomicsbioRxivPreprintDec 23, 2019

Topologically Dependent Abundance of Spontaneous DNA Damage in Single Human Cells

Qiangyuan Zhu, Yichi Niu, Michael C. Gundry…·Chenghang Chuck Zong

This paper introduces a new single-cell WGA method to detect DNA damage-associated variants and finds that dSNV abundance is linked to genome topological compartments.

2 citations

humanwhole-genome sequencing

𝕏 PostBluesky