Mathilde Grelon

This study shows that Arabidopsis thaliana meiotic centromeres undergo rapid movements during zygotene and pachytene stages, which depend on SUN-domain proteins and are disrupted in sun1 sun2 mutants.

The paper investigates how meiotic crossover patterning is controlled by ZMM proteins and the synaptonemal complex, showing that HEI10 overexpression increases crossovers and reduces interference while maintaining sexual dimorphism, and ZYP1 mutation disrupts the link between axis length and COs.

The paper identifies FLIP as a novel partner of FIGL1 that forms a conserved complex regulating meiotic homologous recombination in Arabidopsis and humans.