Chloé Girard

This study shows that polymorphism increases recombination rates in Arabidopsis thaliana, shifting crossover hotspots to polymorphic regions and reducing them in non-polymorphic regions, driven by mismatch repair machinery.

This study identifies FIGL1's role in attenuating meiotic interhomolog repair and shows that XRCC2 and ASY1 counteract FIGL1's effects in Arabidopsis.

This study identifies the HAL-2/HAL-3 complex as essential for regulating Polo-like kinase activity during C. elegans meiosis, ensuring proper homolog pairing and synaptonemal complex assembly.

This study identifies NBS-1 as essential for processing meiotic DSBs to promote crossover formation and antagonize error-prone repair, ensuring genome integrity.

The paper identifies FLIP as a novel partner of FIGL1 that forms a conserved complex regulating meiotic homologous recombination in Arabidopsis and humans.