Author

Ken Natsuga

Hokkaido UniversityORCID 0000-0003-3865-6366Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 2 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Aiko SadaiD· 1 shared Asm Sakhawat HossainiD· 1 shared Erna RajaiD· 1 shared Hideki Nakamura· 1 shared Hideyuki UjiieiD· 1 shared Hiromi YanagisawaiD· 1 shared Hiroshi ShimizuiD· 1 shared Hiroyuki KitahataiD· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperCell Biology

Aiko SadaORCID

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3 papers

Recent·Most cited

Cell BiologyCellular and Molecular Life SciencesPeer-reviewedOct 31, 2026

The epidermal stem cell-supporting matricellular protein fibulin 7 modulates skin inflammatory response in a psoriasis model

Erna Raja, Tomoko Machida, Narenmandula Narenmandula…·Hiromi Yanagisawa

This study shows fibulin 7 protects against skin inflammation by modulating IL-17A signaling in a psoriasis model.

DOI 10.1007/s00018-026-06511-8

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Cell BiologybioRxivPreprintNov 12, 2024

Spatial confinement induces oscillatory migration of epidermal keratinocytes and forms a three-compartment epithelial structure with epithelial-mesenchymal transition dynamics

Takuma Nohara, Sota Itamoto, Junichi Kumamoto…·Ken Natsuga

This study shows that keratinocytes migrate through confined spaces in an oscillatory manner, forming a three-compartment epithelial structure with EMT and MET dynamics.

DOI 10.1101/2024.11.12.623158

𝕏 PostBluesky

Molecular BiologybioRxivPreprintAug 19, 2020

Curing genetic skin disease through altered replication stress response

T. Miyauchi, Shotaro Suzuki, Masae Takeda…·Toshifumi Nomura

The paper shows that mutant CARD14 proteins induce DNA double-strand breaks under replication stress, leading to homologous recombination and reversion of pathogenic mutations in genetic skin diseases.

1 citation

humanDNA methylation

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