Safwan K. Elkhatib

This study shows that traumatic stress increases blood pressure sensitivity to angiotensin II via T-lymphocytes, independent of psychopathology, using a mouse model of repeated social defeat stress.

This study shows that beta-adrenergic signaling and T-lymphocyte-produced catecholamines are required for IL-17A synthesis, offering new therapeutic targets for PTSD and autoimmune diseases.