Author

Jeffrey D. Steinberg

University of California San DiegoORCID 0000-0003-4110-3586Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Genomics· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Assay:bulk RNA-seq· 2 Assay:ATAC-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:machine learning (classical)· 1

Frequent Co-Authors

Cassandra Bui· 2 shared Eduardo Frías-AnayaiD· 2 shared Helios Gallego-GutiérreziD· 2 shared JoAnn TrejoiD· 2 shared Miguel Alejandro Lopez‐RamireziD· 2 shared Aaryaman Sawhney· 1 shared Andrea Taddei· 1 shared Andrew Jackson MurphyiD· 1 shared

Activity by Year

2024-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersPathology

Cassandra Bui

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

PathologybioRxivPreprintDec 11, 2025

Integration of artificial intelligence and high-content screening enabled identification of drugs for long-term treatment of cerebral cavernous malformation disease

Eduardo Frías-Anaya, Helios Gallego-Gutiérrez, Cassandra Bui…·Miguel Alejandro Lopez‐Ramirez

This paper identifies metformin as a potential drug for long-term treatment of cerebral cavernous malformations using AI, high-throughput screening, and animal models.

DOI 10.64898/2025.12.08.693036

mousebulk RNA-seq

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GenomicsbioRxivPreprintJun 29, 2025

Persistent Activation of Endothelial Cells is Linked to Thrombosis and Inflammation in Cerebral Cavernous Malformation Disease

Helios Gallego-Gutiérrez, Eduardo Frías-Anaya, Cassandra Bui…·Miguel Alejandro Lopez‐Ramirez

This paper identifies JUNB as a key driver of chronic brain vascular inflammation in CCM disease through multi-omic analysis of endothelial cell activation.

2 citations

mousescRNA-seq

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Cancer BiologyCancer ResearchPeer-reviewedNov 12, 2024

Conditional c-MYC activation in catecholaminergic cells drives distinct neuroendocrine tumors: neuroblastoma vs somatostatinoma

Tingting Wang, Lingling Liu, Jie Fang…·Jun Yang

This study creates two mouse models with conditional c-MYC activation in specific cell types, leading to distinct neuroendocrine tumors resembling human neuroblastoma and somatostatinoma.

6 citations

mouseexperimental assay

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