Author

Sandipkumar Darji

Nathan Kline Institute for Psychiatric ResearchORCID 0000-0003-4197-4971Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Assay:imaging· 1 Organism:mouse· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

Philip H. StavridesiD· 2 shared Ralph A. NixoniD· 2 shared Asok R. Kumar· 1 shared Corrinne M. Rosa· 1 shared Deanna M. MarchioniniiD· 1 shared Dun‐Sheng YangiD· 1 shared Eunju ImiD· 1 shared Hediye Erdjument‐BromageiD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersPathology

Philip H. StavridesORCID

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2 papers

Recent·Most cited

PathologyActa Neuropathologica CommunicationsPeer-reviewedNov 7, 2025

Pathobiology of the autophagy-lysosomal pathway in the Huntington's disease brain

Martin J. Berg, Veeranna, Corrinne M. Rosa…·Ralph A. Nixon

This study investigates the role of the autophagy-lysosomal pathway in Huntington's disease, finding that autophagic flux declines due to substrate clearance deficits in later stages.

15 citations

humanimaging

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NeuroscienceScience AdvancesPeer-reviewedJul 26, 2023

Lysosomal dysfunction in Down Syndrome and Alzheimer mouse models is caused by selective v-ATPase inhibition by Tyr682 phosphorylated APP βCTF

Eunju Im, Ying Jiang, Philip H. Stavrides…·Ralph A. Nixon

This study identifies that phosphorylated APP βCTF inhibits v-ATPase in Down Syndrome and Alzheimer's models, leading to lysosomal dysfunction.

80 citationstop 1%

humanmouse

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