Author

Ying Jiang

Nathan Kline Institute for Psychiatric ResearchORCID 0000-0003-0219-7807Unclaimed ORCID profile · follow to get notified of new preprints

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Ying Jiang7 papers · Boston University

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Research Focus:protein structure prediction· 1 Assay:proteomics· 1

Frequent Co-Authors

Philip H. StavridesiD· 2 shared Ralph A. NixoniD· 2 shared Anna PensalfiniiD· 1 shared Balapal S. BasavarajappaiD· 1 shared Chris N. GoulbourneiD· 1 shared Cynthia Bleiwas· 1 shared Eunju ImiD· 1 shared Hediye Erdjument‐BromageiD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersNeuroscience

Philip H. StavridesORCID

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2 papers

Recent·Most cited

NeuroscienceJournal of NeurosciencePeer-reviewedJun 13, 2025

Increased neuronal expression of the early endosomal adaptor APPL1 replicates Alzheimer's Disease-related endosomal and synaptic dysfunction with cholinergic neurodegeneration

Ying Jiang, Kuldeep Sachdeva, Chris N. Goulbourne…·Ralph A. Nixon

This study shows that overexpressing APPL1 in neurons causes AD-like endosomal and synaptic dysfunction, cholinergic neurodegeneration, and memory deficits in mice.

2 citations

mouseexperimental assay

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NeuroscienceScience AdvancesPeer-reviewedJul 26, 2023

Lysosomal dysfunction in Down Syndrome and Alzheimer mouse models is caused by selective v-ATPase inhibition by Tyr682 phosphorylated APP βCTF

Eunju Im, Ying Jiang, Philip H. Stavrides…·Ralph A. Nixon

This study identifies that phosphorylated APP βCTF inhibits v-ATPase in Down Syndrome and Alzheimer's models, leading to lysosomal dysfunction.

80 citationstop 1%

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