Author

Ritchie Williamson

University of BradfordORCID 0000-0003-4278-0909Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:proteomics· 2 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Samantha Louise McLeaniD· 2 shared Andrew D. RandalliD· 1 shared Andrew ParnelliD· 1 shared Ben Mellows· 1 shared Craig BealliD· 1 shared Daniel Feist· 1 shared John K. ChiltoniD· 1 shared Josephine Louise RobbiD· 1 shared

Activity by Year

2018-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersNeuroscience

Samantha Louise McLeanORCID

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3 papers

Recent·Most cited

NeurosciencebioRxivPreprintJul 4, 2026

ESCRT Machinery Dysfunction in Motor Neurone Disease: TSG101, CHMP2B, and VPS4a Differentially Regulate TDP-43 Pathology, Autophagy, and Exosome Biogenesis

Laila A.A.H. Mohamed, Mohamed Fouad Shalaby, Ritchie Williamson…·Sriharsha Kantamneni

DOI 10.64898/2026.07.01.735805

humanproteomics

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NeurosciencebioRxivPreprintMar 20, 2026

A stem cell secretome delays functional decline and supresses inflammation in two distinct models of neurodegeneration.

S. Dickens, Andrew Parnell, Daniel Feist…·Ritchie Williamson

This study shows that stem cell secretomes improve behavioral and motor function in two neurodegenerative models and reduce inflammation without altering neuropathology.

DOI 10.64898/2026.03.18.712006

𝕏 PostBluesky

NeurosciencebioRxivPreprintFeb 26, 2018

Human primary astrocytes increase basal fatty acid oxidation following recurrent low glucose to maintain intracellular nucleotide levels

Paul G. Weightman Potter, Julia M. Vlachaki Walker, Josephine Louise Robb…·Craig Beall

This study shows that human astrocytes increase fatty acid oxidation under recurrent low glucose to maintain nucleotide levels, indicating mitochondrial dysfunction in hypoglycemia response.

1 citation

humanmetabolomics

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