Author

Hideo Yagita∥

Juntendo UniversityORCID 0000-0003-4279-9343Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

Atsumi Tsuji‐HosokawaiD· 1 shared Chiyono Morimoto· 1 shared Hiroshi OhnoiD· 1 shared Hitomi Suzuki· 1 shared Josephine BowlesiD· 1 shared Kenichi KashimadaiD· 1 shared Kiyo‐aki IshiiiD· 1 shared Koji HaseiD· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperDevelopmental Biology

Atsumi Tsuji‐HosokawaORCID

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2 papers

Recent·Most cited

MicrobiologybioRxivPreprintJun 26, 2026

LRP1 is an entry receptor for the botulinum toxin complex in the gut

Sho Amatsu, Takuhiro Matsumura, Chiyono Morimoto…·Yukako Fujinaga

This study identifies LRP1 as the receptor for L-PTC/BOkra to enter the gut, showing HA binds to LRP1 via N-glycans and facilitates transcytosis.

DOI 10.64898/2026.06.25.734645

mouseproteomics

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Developmental BiologyJournal of Cell SciencePeer-reviewedMar 15, 2019

Nr5a1 suppression during the fetal period optimizes ovarian development by fine-tuning of Notch signaling

Risa Nomura, Kenichi Kashimada, Hitomi Suzuki…·Tomohiro Morio

This paper shows that Nr5a1 suppression during fetal development optimizes ovarian development by fine-tuning Notch signaling, using mouse models with altered Nr5a1 expression.

6 citations

mouseexperimental assay

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