Jing Wang

The paper identifies a developmental timer in C. elegans that coordinates microRNA transcription across tissues through a feedback loop between MYRF-1 and LIN-42.

This study investigates the impact of substrate stiffness on C. elegans health and longevity, finding only mild effects on cellular and organismal health.

The paper shows that NHR-85 and NHR-23 heterodimers regulate lin-4 miRNA transcription pulses in C. elegans, similar to circadian clock mechanisms in mammals.

This paper identifies pqn-59 as a factor that antagonizes miRNA-mediated repression and promotes stress granule formation in C. elegans, contributing to regulatory bistability in developmental gene networks.