Author

Sheng‐Yang Ho

University of California, DavisORCID 0009-0000-2292-6494Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

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Assay:electronic health records· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:protein structure prediction· 1 Approach:statistical model· 1

Frequent Co-Authors

Johannes Wilhelm HelliD· 2 shared Madeline Nieves‐CintróniD· 2 shared Manuel F. NavedoiD· 2 shared Mary C. HorneiD· 2 shared Peter H. BartelsiD· 2 shared Andranik Avedisyan· 1 shared Ariel A. Jacobi· 1 shared Chao-Yin CheniD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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2 shared papersNeuroscience

Johannes Wilhelm HellORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintApr 29, 2026

A Unifying Mechanism for Synaptic Amyloid beta Toxicity Beta Adrenergic Potentiation of the Ca2+ Channel CaV1.2 by Amyloid beta

Peter H. Bartels, Sarah L. S. Rougé, James D. Scripter…·Johannes W. Hell

This paper identifies a mechanism by which amyloid beta potentiates CaV1.2 channels via β2AR, leading to synaptic dysfunction and neurotoxicity, and shows that disrupting this pathway prevents these effects.

1 citation

mouseelectronic health records

𝕏 PostBluesky

NeuroscienceeLifePeer-reviewedJun 20, 2023

α1 adrenergic receptor - PKC - Pyk2 - Src signaling boosts L-type Ca2+ channel Cav1.2 activity and long-term potentiation in rodents

Kwun Nok Mimi Man, Peter Bartels, Peter B. Henderson…·Johannes Wilhelm Hell

This study identifies the α1 adrenergic receptor - PKC - Pyk2 - Src signaling pathway as a mechanism boosting CaV1.2 channel activity and LTP in rodents.

13 citations

mouseelectronic health records

𝕏 PostBluesky