Author

Julius I. Yevdash

University of IowaORCID 0009-0001-6482-2279Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Colin KennyiD· 2 shared Collin Montgomery· 2 shared Katelyn R. Campbell-HansoniD· 2 shared Robert A. CornelliD· 2 shared Ronald J. WeigeliD· 2 shared Adam J. DupuyiD· 1 shared Alexander BirbrairiD· 1 shared Annika HelversoniD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersCancer Biology

Colin KennyORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Cancer BiologyCancer ResearchPeer-reviewedMay 26, 2026

Mitfa-Independent Melanocyte Progenitors are Highly Susceptible to GNAQ-induced Uveal Melanoma in Adult Zebrafish

Julius I. Yevdash, Delaney Robinson, Rachel Moore…·Colin Kenny

This study shows that Mitfa-independent melanocyte progenitors in zebrafish are more susceptible to GNAQ-induced uveal melanoma than differentiated melanocytes.

1 citationtop 4%

zebrafishscRNA-seq

𝕏 PostBluesky

Cancer BiologyCell ReportsPeer-reviewedMar 26, 2025

Antagonistic Roles for MITF and TFE3 in Melanoma Plasticity

Jeremy Chang, Katelyn R. Campbell-Hanson, Marion Vanneste…·Colin Kenny

This study identifies TFE3 and the non-canonical mTORC1 pathway as regulators of melanoma cell state plasticity, showing MITF suppresses the mesenchymal phenotype by modulating TFE3 expression.

11 citations

humanbulk RNA-seq

𝕏 PostBluesky