Author

Ronald J. Weigel

University of IowaORCID 0000-0002-9249-0793Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1

Frequent Co-Authors

Colin KennyiD· 2 shared Collin Montgomery· 2 shared Julius I. YevdashiD· 2 shared Katelyn R. Campbell-HansoniD· 2 shared Robert A. CornelliD· 2 shared Adam J. DupuyiD· 1 shared Alexander BirbrairiD· 1 shared Allison W. Lorenzen· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

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2 shared papersCancer Biology

Colin KennyORCID

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Author profile →

3 papers

Recent·Most cited

Mitfa-Independent Melanocyte Progenitors are Highly Susceptible to GNAQ-induced Uveal Melanoma in Adult Zebrafish

Julius I. Yevdash, Delaney Robinson, Rachel Moore, Zhijie Li, Katelyn R. Campbell-Hanson, Danielle Gutelius, Stephen P.G. Moore, Dylan Friend, Isaac O'Toole, Brynnon E. Harman, Collin Montgomery, Robert A. Cornell, Alexander Birbrair, Jesse D. Riordan, Adam J. Dupuy, , Colin Kenny

This study shows that Mitfa-independent melanocyte progenitors in zebrafish are more susceptible to GNAQ-induced uveal melanoma than differentiated melanocytes.

Cancer Research · May 26, 2026Peer-reviewedCancer Biology

Antagonistic Roles for MITF and TFE3 in Melanoma Plasticity

Jeremy Chang, Katelyn R. Campbell-Hanson, Marion Vanneste, Nicholas I. Bartschat, R. H. Nagel, Asdis K Arnadottir, Hong Nhung Vu, Collin Montgomery, Julius I. Yevdash, Jiarui Jiang, Ardith Bhinu, Annika Helverson, Michael D. Henry, Eirı́kur Steingrı́msson, Ronald J. Weigel, , Colin Kenny

This study identifies TFE3 and the non-canonical mTORC1 pathway as regulators of melanoma cell state plasticity, showing MITF suppresses the mesenchymal phenotype by modulating TFE3 expression.

Cell Reports · Mar 26, 2025Peer-reviewedCancer Biology

AP-2γ is Required for Maintenance of Pluripotent Mammary Stem Cells

Vivian W. Gu, Edward Cho, Dakota T. Thompson, Victoria C. Cassady, Nicholas C. Borcherding, Kelsey E. Koch, Vincent T. Wu, Allison W. Lorenzen, Dana M. van der Heide, Jeffrey R. White, Mikhail V. Kulak, Trevor Williams, Weizhou Zhang, Ronald J. Weigel

This study shows that AP-2γ is essential for maintaining pluripotent mammary stem cells and their ability to form mammary gland structures.

Stem Cell Reports · Dec 30, 2020Peer-reviewedDevelopmental Biology