Author

Almudena Moreno-Rivero

Universidad de SevillaORCID 0009-0001-8825-8473Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:C. elegans· 2 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

Alejandro Mata‐CabanaiD· 2 shared María OlmedoiD· 2 shared Marta Muñoz-BarreraiD· 2 shared Beatriz Ren-BarrosoiD· 1 shared Daphne S. CabiancaiD· 1 shared Francesca SartoriD· 1 shared Francine PiubeliiD· 1 shared Francisco Javier Romero-ExpósitoiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersDevelopmental Biology

Alejandro Mata‐CabanaORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Developmental BiologyNucleic Acids ResearchPeer-reviewedSep 21, 2026

HLH-30/TFEB is necessary for chromatin reorganization and maintenance of cell quiescence during starvation in C. elegans

Marta Muñoz-Barrera, Alejandro Mata‐Cabana, Almudena Moreno-Rivero…·María Olmedo

This study shows HLH-30/TFEB is essential for chromatin reorganization and cell quiescence during starvation in C. elegans, with loss of HLH-30 disrupting cell cycle arrest and causing premature developmental activation.

1 citation

C. elegansChIP-seq

𝕏 PostBluesky

Developmental BiologybioRxivPreprintMar 31, 2024

Desynchrony between events triggers a compensatory delay during C. elegans development

Francisco Javier Romero-Expósito, Almudena Moreno-Rivero, Marta Muñoz-Barrera…·María Olmedo

The paper shows that reduced insulin signaling in C. elegans delays cell divisions more than molting, increasing desynchrony and triggering a compensatory delay in the molting program.

1 citation

C. elegansexperimental assay

𝕏 PostBluesky