Alejandro Mata‐Cabana

HLH-30/TFEB is necessary for chromatin reorganization and maintenance of cell quiescence during starvation in C. elegans

This study shows HLH-30/TFEB is essential for chromatin reorganization and cell quiescence during starvation in C. elegans, with loss of HLH-30 disrupting cell cycle arrest and causing premature developmental activation.

Desynchrony between events triggers a compensatory delay during C. elegans development

The paper shows that reduced insulin signaling in C. elegans delays cell divisions more than molting, increasing desynchrony and triggering a compensatory delay in the molting program.

Differential regulation of developmental stages supports a linear model for C. elegans postembryonic development

The paper measures developmental stage durations in C. elegans under varied conditions and finds that stages respond differently to environmental factors, supporting a linear progression model.

The cellular modifier MOAG-4/SERF drives amyloid formation through charge complementation

The paper identifies MOAG-4/SERF as modifiers of amyloid formation through charge complementation, showing that charge interactions drive protein aggregation and toxicity.

Neuronal perception of the social environment intergenerationally controls germline development and generation time in C. elegans

The paper shows that neuronal perception of social pheromones in C. elegans affects germline development timing and generation time in offspring.

Social chemical communication determines recovery from L1 arrest via DAF-16 activation

This study shows that social chemical communication via trehalose and DAF-16 activation enhances recovery from L1 arrest in C. elegans.

Prolonged quiescence delays somatic stem cell-like division in Caenorhabditis elegans and is controlled by insulin signalling

This study shows prolonged quiescence in C. elegans L1 larvae delays cell division and is regulated by insulin signaling, with maternal age affecting recovery variability.