Author

Berenice Fischer

Johannes Gutenberg University MainzORCID 0009-0004-1330-7156Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

Ana-Marija Kulis-Mandic· 1 shared Antonia KolbiD· 1 shared Ari WaismaniD· 1 shared Daniela KrameriD· 1 shared Delia Mihaela Mihoc· 1 shared Emmanouil StylianakisiD· 1 shared Kartikeya SinghiD· 1 shared Klaus Schulze‐OsthoffiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperImmunology

Ana-Marija Kulis-Mandic

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1 paper

Recent·Most cited

ImmunologybioRxivPreprintFeb 24, 2026

Treg-derived IκBζ promotes their conversion into Th2-like effectors and drives type 2 inflammation via BATF

Tanja Kübelbeck, Ana-Marija Kulis-Mandic, Antonia Kolb…·Daniela Kramer

This study identifies IκBζ as a key regulator of Treg cell plasticity, showing it drives their conversion into Th2-like effectors and promotes type 2 inflammation.

DOI 10.64898/2026.02.23.707402

mousegenetic perturbation / CRISPR

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