Author

Antonia Kolb

Johannes Gutenberg University MainzORCID 0009-0001-2790-9551Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:host-pathogen & immune response· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Ana-Marija Kulis-Mandic· 2 shared Daniela KrameriD· 2 shared Klaus Schulze‐OsthoffiD· 2 shared Matthias KleiniD· 2 shared Alpaslan TasdoganiD· 1 shared Andrea TuettenbergiD· 1 shared Anna Stastny· 1 shared Antje SuckeriD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersImmunology

Ana-Marija Kulis-Mandic

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2 papers

Recent·Most cited

ImmunologybioRxivPreprintFeb 24, 2026

Treg-derived IκBζ promotes their conversion into Th2-like effectors and drives type 2 inflammation via BATF

Tanja Kübelbeck, Ana-Marija Kulis-Mandic, Antonia Kolb…·Daniela Kramer

This study identifies IκBζ as a key regulator of Treg cell plasticity, showing it drives their conversion into Th2-like effectors and promotes type 2 inflammation.

DOI 10.64898/2026.02.23.707402

mousegenetic perturbation / CRISPR

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Cancer BiologyNature CommunicationsPeer-reviewedJun 25, 2025

Constitutive expression of IκBζ promotes tumor growth and immunotherapy resistance in melanoma

Antonia Kolb, Ana-Marija Kulis-Mandic, Matthias Klein…·Daniela Kramer

This study shows that constitutive IκBζ expression in melanoma promotes tumor growth and immunotherapy resistance by suppressing cytokine production and immune cell recruitment.

10 citationstop 5%

humanproteomics

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