Author

Asa Nakahara

Brain Research Institute, Niigata UniversityORCID 0009-0004-2944-4403Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2

Frequent Co-Authors

Akiyoshi KakitaiD· 2 shared A. SakaiiD· 1 shared Ai ItoiD· 1 shared Alexey G. Murzin· 1 shared Atsushi HatanoiD· 1 shared Benjamin FalconiD· 1 shared Bert M. VerheijeniD· 1 shared Chao QiiD· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersNeuroscience

Akiyoshi KakitaORCID

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2 papers

Recent·Most cited

Extracellular disposal of nuclear waste by APP: a protective mechanism impaired in Alzheimer's disease

Godfried Dougnon, Takayoshi Otsuka, Yuka Nakamura, A. Sakai, Tomoyuki Yamanaka, Noriko Matsui, Asa Nakahara, Ai Ito, Atsushi Hatano, Masaki Matsumoto, Hironaka Igarashi, Akiyoshi Kakita, Masaki Ueno, Hideaki Matsui

This study identifies APP's role in extracellular disposal of nuclear waste, showing its impairment in Alzheimer's disease and suggesting it as a therapeutic target.

bioRxiv · Feb 10, 2024 · PreprintNeuroscience

Tau Filaments from Amyotrophic Lateral Sclerosis/Parkinsonism-Dementia Complex (ALS/PDC) adopt the CTE Fold

Chao Qi, Bert M. Verheijen, Yasumasa Kokubo, Yang Shi, Stephan Tetter, Alexey G. Murzin, Asa Nakahara, Satoru Morimoto, Marc Vermulst, Ryogen Sasaki, Eleonora Aronica, YOSHIFUMI S. HIROKAWA, Kiyomitsu Oyanagi, Akiyoshi Kakita, Benjamin Falcon, , Michel Goedert

This study uses cryo-EM to show that tau filaments in ALS/PDC share the CTE fold, suggesting environmental causes for the disease.

bioRxiv · Apr 26, 2023 · PreprintNeuroscience