Cancer BiologyCancer ResearchPeer-reviewed
Endocrine persistence in ER+ breast cancer is accompanied by metabolic vulnerability in oxidative phosphorylation
Steven S. Tau, Mary Dickinson Chamberlin, Huijuan Yang…·Todd W. Miller
This study identifies mitochondrial oxidative phosphorylation as a metabolic vulnerability in endocrine-tolerant persister ER+ breast cancer cells, suggesting therapeutic targeting of mitochondrial function could prevent recurrence.
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