Author

Prerna Chahal

Michigan State UniversityORCID 0009-0004-4770-0853Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Abigail Z. BennettiD· 1 shared Andrew J. OliveiD· 1 shared Caitlin S. WilliamsoniD· 1 shared Canchai YangiD· 1 shared Craig C. WelboniD· 1 shared Dara M VillaiD· 1 shared Dohun PyeoniD· 1 shared Evelyn Gomez RecinosiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

Abigail Z. BennettORCID

Mutual co-authors in cluster

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1 paper

Recent·Most cited

ULK1 drives NDP52-mediated selective autophagic degradation of MHC-I to promote immune evasion in HPV-positive head and neck cancer

Lexi Vu, Nicholas S. Giacobbi, M. I. Khalil, Canchai Yang, Will Eckerman, Evelyn Gomez Recinos, Johnathon D. Garber, Hyomin Son, Prerna Chahal, Dara M Villa, Tasha Srivastava, Abigail Z. Bennett, Katie R. Martin, Craig C. Welbon, Caitlin S. Williamson, , Dohun Pyeon

This study identifies ULK1-driven autophagy as a mechanism by which HPV+ HNC cells degrade MHC-I, promoting immune evasion and tumor growth.

bioRxiv · Mar 18, 2026 · PreprintCancer Biology