Author

Vanessa Nomellini

The University of Texas Southwestern Medical CenterORCID 0009-0004-6284-5006Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Bioengineering· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Organism:human· 1 Assay:metabolomics· 1

Frequent Co-Authors

Alexander M. TataraiD· 1 shared Angela J. RogersiD· 1 shared Ankita Sarkar· 1 shared Anoj IlangesiD· 1 shared Ashutosh ShuklaiD· 1 shared Bhaskar VundurthyiD· 1 shared Caroline N. JonesiD· 1 shared Carolyn S. CalfeeiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperPhysiology

Alexander M. TataraORCID

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2 papers

Recent·Most cited

Lipid mobilization establishes metabolic tolerance and prevents autonomic collapse in infection

Ankita Sarkar, Shangkui Xie, Syed Kazim Rizvi, Tadiwanashe Gwatiringa, Sophie Heston, Samuel Piaker, Narges Alipanah, Jianyi Yin, Laurent Gautron, Soumya Kamath, Neena Alex, Ashutosh Shukla, Lin Jia, Rani Shiao, Lauren E Kemp, , Suraj J. Patel

This study identifies white adipose tissue lipolysis and circulating fatty acids as key mediators of metabolic tolerance to infection, showing improved survival in sepsis with beta3-adrenergic agonists.

bioRxiv · Apr 21, 2026 · PreprintPhysiology

Murine Neutrophil Chemotaxis Following Burn Injury with Poloxamer 188 Treatment in a Microfluidic Platform

Hossein Razmi Bagtash, Nagham Alatrash, Udaya Sree Datla, Bhaskar Vundurthy, Shuai Shao, Roopa Koduri, Mecca B.A.R. Islam, Kevin T. Mutore, Emma Salari, Renqing Wu, Vanessa Nomellini, Caroline N. Jones

This study shows that Poloxamer 188 increases neutrophil migration toward LTB4 in both male and female mice after burn injury using a microfluidic platform.

bioRxiv · Jan 8, 2026 · PreprintBioengineering