Kendal F. Willcox

The paper shows that B cell-derived IgG in DRG meninges forms tertiary lymphoid structures (TLSs) that drive neuropathic pain after peripheral nerve injury, and disrupting TLSs prevents pain.

This study shows that B cell-derived autoreactive IgG and FcγR upregulation drive mechanical allodynia after nerve constriction or crush injury, but not after nerve transection.

This study shows that voluntary wheel running before sciatic nerve injury reduces neuropathic pain in rats by enhancing antioxidant defenses through Nrf2 signaling.