Author

Antonios Chronopoulos

Children's Hospital Los AngelesORCID 0009-0006-1197-2341Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cell Biology· 2 Pathology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1 Assay:imaging· 1

Frequent Co-Authors

Alexandre de Fraipont· 1 shared Aman YarlagaddaiD· 1 shared Andrew Richardson· 1 shared Barbara A. Moreno Diaz· 1 shared Chandra Kaladhar VemulaiD· 1 shared Gregory W. CharvilleiD· 1 shared Hikaru SugimotoiD· 1 shared Ivan ChaveziD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperPathology

Alexandre de Fraipont

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Cell BiologybioRxivPreprintAug 11, 2026

Engineered extracellular vesicles targeting BACE1 reduces amyloid beta plaque formation in a genetic mouse model of Alzheimer Disease

Valerie S. Kalluri, Sara P.Y. Che, Meagan Conner…·Raghu Kalluri

This study shows engineered extracellular vesicles delivering BACE1-targeting siRNA reduce amyloid beta plaques in a mouse model of Alzheimer's disease.

DOI 10.64898/2026.08.10.744066

mouseproteomics

𝕏 PostBluesky

PathologybioRxivPreprintJul 24, 2026

Hyaluronan and CD44 targeting reverses early matrix changes, proinflammatory signals and fibrosis in primary sclerosing cholangitis

Vidushi Bansal, Lóránd Váncza, Weiguo Fan…·Natalie Julia Torok

This study identifies HA and CD44 as mediators of matrix changes and fibrosis in PSC, showing their targeting can reverse disease progression.

DOI 10.64898/2026.07.21.739845

humanbulk RNA-seq

𝕏 PostBluesky

Cell BiologyThe Journal of Cell BiologyPeer-reviewedApr 22, 2025

Differential cell-ECM interaction of rhabdomyosarcoma subtypes regulated by PAX3-FOXO1

Antonios Chronopoulos, Ivan Chavez, Chandra Kaladhar Vemula…·JinSeok Park

This study identifies differences in cell-ECM interaction between rhabdomyosarcoma subtypes, showing FNRMS has heightened interaction regulated by TGFβ signaling, while PAX3-FOXO1 in FPRMS suppresses this interaction.

1 citation

humanscRNA-seq

𝕏 PostBluesky