Author

Yingzheng Wang

Cornell UniversityORCID 0009-0006-1570-3624Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biochemistry· 1 Molecular Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:chromatin & epigenetic regulation· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Khoula Jaber· 2 shared Marcus Bustamante SmolkaiD· 2 shared Deanna V. MaybeeiD· 1 shared Eliz Çelik· 1 shared Jiyang ZhangiD· 1 shared Jumana BadariD· 1 shared M.V.A.S. NavarroiD· 1 shared Mateusz Maciej WagneriD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersMolecular Biology

Khoula Jaber

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Pharmacology & ToxicologybioRxivPreprintMay 1, 2026

Inhibition of oogenic JNK preserves fertility and ovarian hormones during DNA-damaging cancer therapy

Wenlong Zhao, Jiyang Zhang, Yingnan Bo…·Shuo Xiao

This study shows that inhibiting JNK in oocytes protects against chemotherapy-induced infertility and ovarian hormone loss in mice.

DOI 10.64898/2026.04.28.721450

mouseexperimental assay

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Molecular BiologyNature CommunicationsPeer-reviewedNov 13, 2025

Proteomic Sensors for Quantitative, Multiplexed and Spatial Monitoring of Kinase Signaling

William J. Comstock, M.V.A.S. Navarro, Deanna V. Maybee…·Marcus Bustamante Smolka

This paper introduces ProKAS, a proteomic platform for spatial and quantitative analysis of kinase activity using mass spectrometry, and demonstrates its application in studying DNA damage response kinases.

2 citations

proteomicsexperimental assay

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BiochemistrybioRxivPreprintMay 13, 2025

An ATM-PPM1D Circuit Controls the Processing and Restart of DNA Replication Forks

Yiting Cao, Yingzheng Wang, Jumana Badar…·Marcus Bustamante Smolka

This study identifies PPM1D as a regulator of replication fork restart, showing it prevents excessive nucleolytic degradation and facilitates RAD51-dependent restart, with ATM hyper-signaling counteracting PPM1D loss.

2 citations

humanstatistical model

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