Author

Huilan Wang

Fudan UniversityORCID 0009-0008-4452-489XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Organism:Drosophila· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Boxun LuiD· 2 shared Alma M. PereziD· 1 shared Andrew Laitman· 1 shared Bingjie ZhangiD· 1 shared Genevera I. AlleniD· 1 shared Guang YangiD· 1 shared He ZhaoiD· 1 shared Hye Min KimiD· 1 shared

Activity by Year

2020-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

Boxun LuORCID

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2 papers

Recent·Most cited

Cell BiologyNature Chemical BiologyPeer-reviewedMar 14, 2025

Post-translational modifications orchestrate the intrinsic signaling bias of orphan receptor GPR52

Bingjie Zhang, Wei Ge, Mengna Ma…·Wenqing Shui

This study identifies how N-glycosylation and phosphorylation in specific motifs of GPR52 differentially regulate its signaling bias and cellular function.

3 citations

humanproteomics

𝕏 PostBluesky

Systems BiologybioRxivPreprintNov 3, 2020

Modulating glial genes involved in synaptic function mitigates pathogenesis and behavioral deficits in a Drosophila model of Huntington's Disease

Tarik Seref Onur, Andrew Laitman, He Zhao…·Juan Botas

The paper shows that reducing glial genes involved in synaptic function reduces disease pathology and behavioral issues in a Drosophila Huntington's Disease model.

DOI 10.1101/2020.11.03.367326

Drosophilabulk RNA-seq

𝕏 PostBluesky