Author

A. Ciliberto

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:chromatin & epigenetic regulation· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Approach:statistical model· 1 Assay:whole-genome sequencing· 1 Organism:yeast· 1

Frequent Co-Authors

F. Gross· 2 shared A. Poti· 1 shared A. T. Saurin· 1 shared D. Szuts· 1 shared E. Chiroli· 1 shared F. Macaluso· 1 shared F. Natali· 1 shared G. Rancati· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

F. Gross

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2 papers

Recent·Most cited

Epistasis, aneuploidy, and gain-of-function mutations underlie the evolution ofresistance to induced microtubule depolymerization

M. Pavani, P. Bonaiuti, E. Chiroli, F. Gross, F. Natali, F. Macaluso, A. Poti, S. Pasqualato, S. Pompei, M. Cosentino Lagomarsino, G. Rancati, D. Szuts, A. Ciliberto

The paper evolves yeast populations with impaired microtubule polymerization and identifies mutations and chromosome duplications that confer resistance to microtubule depolymerizing drugs.

bioRxiv · Oct 7, 2020 · PreprintCell Biology

PP1 and PP2A use opposite phospho-dependencies to control distinct processes at the kinetochore

R. J. Smith, M. H. Cordeiro, N. E. Davey, G. Vallardi, A. Ciliberto, F. Gross, A. T. Saurin

The paper shows PP1 and PP2A-B56 use opposite phospho-dependencies to control distinct mitotic processes at the kinetochore through differential kinase input responses.

bioRxiv · May 5, 2019Cell Biology