Author

A. D'Amico

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1 Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 5 Organism:human· 5 Assay:bulk RNA-seq· 3 Assay:proteomics· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:statistical model· 2

Frequent Co-Authors

A. Bordin· 2 shared E. De Falco· 2 shared F. Pagano· 2 shared G. Frati· 2 shared I. Chimenti· 2 shared V. Picchio· 2 shared A. Bongiovanni· 1 shared A. Cao· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersPathology

A. Bordin

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Human light meromyosin mutations linked to skeletal myopathies disrupt the coiled coil structure and myosin head sequestration

G. Carrington, A. Hau, S. Kosta, H. Dugdale, F. Muntoni, A. D'Amico, P. Van den Bergh, N. Romero, E. Malfatti, J. J. Vilchez, A. Oldfors, S. Pajusalu, K. Ounap, M. Giralt-Pujol, E. Zanoteli, , J. Ochala

This study investigates how mutations in MYH7 and MYH2 genes affect myosin structure and function, finding that they disrupt coiled-coil structure and increase ATP consumption without affecting contractile function.

bioRxiv · May 15, 2023 · PreprintPhysiology

The Fatty Acid Binding Protein Family Represents a Novel Target in Multiple Myeloma

M. Farrell, H. Fairfield, M. Karam, A. D'Amico, C. S. Murphy, C. Falank, R. S. Pistofidi, A. Cao, C. R. Marinac, J. A. Dragon, L. McGuinness, C. G. Gartner, R. D. Iorio, E. Jachimowicz, V. DeMambro, , M. R. Reagan

This paper identifies the fatty acid binding protein (FABP) family as a novel therapeutic target in multiple myeloma, showing that FABP inhibition reduces tumor growth and alters key cellular processes.

eLife · Mar 7, 2023Peer-reviewedCancer Biology

Investigating the risk of cardiac fibrosis due to heat-not-burn cigarettes through human cardiac stromal cells

I. Chimenti, V. Picchio, F. Pagano, R. Carnevale, A. D'Amico, C. Cozzolino, E. Floris, A. Bordin, L. Schirone, w. saade, F. Miraldi, E. De Falco, S. Sciarretta, M. Peruzzi, G. Biondi-Zoccai, G. Frati

This study examines how serum from chronic heat-not-burn cigarette smokers affects cardiac stromal cells, showing increased fibrosis and reduced paracrine support.

bioRxiv · Sep 6, 2022 · PreprintPathology

Human platelet lysate derived extracellular vesicles enhance angiogenesis through miR-126

E. De Falco, A. Bordin, M. Chirivi, F. Pagano, M. Milan, M. Iuliano, E. Scaccia, O. Fortunato, G. Mangino, X. Dhori, E. De Marinis, A. D'Amico, S. Miglietta, V. Picchio, R. Rizzi, , G. Frati

This study shows that platelet lysate-derived extracellular vesicles enhance angiogenesis via miR-126, without relying on VEGF.

bioRxiv · May 10, 2022Cell Biology

Pharmacological tuning of microRNA in FAP-derived Extracellular Vesicles by HDAC inhibitors promotes regeneration and reduces fibrosis in dystrophic muscles.

M. Sandona, S. Consalvi, L. Tucciarone, M. De Bardi, M. Scimeca, D. F. Angelini, V. Buffa, A. D'Amico, E. S. Bertini, S. Cazzaniga, P. Bettica, M. Bouche, A. Bongiovanni, P. L. Puri, V. Saccone

The paper shows that HDAC inhibitors increase microRNA levels in FAP-derived EVs, enhancing muscle regeneration and reducing fibrosis in dystrophic models.

bioRxiv · Feb 15, 2020 · PreprintCell Biology