Author

M. De Bardi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

L. Tucciarone· 2 shared P. L. Puri· 2 shared S. Consalvi· 2 shared A. Bongiovanni· 1 shared A. D'Amico· 1 shared A. Mai· 1 shared A. Renzini· 1 shared C. Cirotti· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersPharmacology & Toxicology

L. Tucciarone

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Author profile →

3 papers

Recent·Most cited

NRF2 connects Src tyrosine kinase to ferroptosis resistance in glioblastoma

D. Barila, C. Cirotti, I. Taddei, C. Contadini, G. Pepe, M. De Bardi, G. Borsellino, M. Helmer-Citterich

This study identifies a molecular link between Src tyrosine kinase and ferroptosis resistance in glioblastoma through NRF2 pathway activation, suggesting a potential therapeutic target.

bioRxiv · May 8, 2023Cancer Biology

Partial resistance to HDAC inhibitors in FAPs of dystrophic muscles at late stages of disease is associated to epigenetic and transcriptional features of cellular senescence.

S. Consalvi, L. Tucciarone, E. Macri, M. De Bardi, M. Picozza, I. Salvatori, A. Renzini, S. Valente, A. Mai, V. Moresi, P. L. Puri

This study shows that HDAC inhibitors are less effective in late-stage DMD muscle FAPs due to senescence-related epigenetic changes that resist reversal.

bioRxiv · Apr 26, 2021 · PreprintPharmacology & Toxicology

Pharmacological tuning of microRNA in FAP-derived Extracellular Vesicles by HDAC inhibitors promotes regeneration and reduces fibrosis in dystrophic muscles.

M. Sandona, S. Consalvi, L. Tucciarone, M. De Bardi, M. Scimeca, D. F. Angelini, V. Buffa, A. D'Amico, E. S. Bertini, S. Cazzaniga, P. Bettica, M. Bouche, A. Bongiovanni, P. L. Puri, V. Saccone

The paper shows that HDAC inhibitors increase microRNA levels in FAP-derived EVs, enhancing muscle regeneration and reducing fibrosis in dystrophic models.

bioRxiv · Feb 15, 2020 · PreprintCell Biology