Author

A. F. Rubin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Evolutionary Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Organism:E. coli· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Foster· 1 shared B. Wang· 1 shared C. Chiu· 1 shared C. E. Gonzalez· 1 shared D. Rohm· 1 shared E. Tresoldi· 1 shared E.-Y. Tsou· 1 shared F. J. Cameron· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperImmunology

A. Foster

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2 papers

Recent·Most cited

Proinsulin C-peptide is a major source of HLA-DQ8 restricted HIPs recognized by human Islet-Infiltrating CD4+ T cells

P. Bhattacharjee, M. Pakusch, M. Lacorcia, E. Tresoldi, A. F. Rubin, A. Foster, L. S. King, C. Chiu, T. Kay, J. A. Karas, F. J. Cameron, S. I. Mannering

This study identifies 13 new proinsulin-derived HIPs recognized by human islet-infiltrating CD4+ T cells in T1D, highlighting proinsulin C-peptide's central role in HIP formation.

bioRxiv · May 9, 2024Immunology

Collateral fitness effects of mutations

J. D. Mehlhoff, F. W. Stearns, D. Rohm, B. Wang, E.-Y. Tsou, N. Dutta, M.-H. Hsiao, C. E. Gonzalez, A. F. Rubin, M. Ostermeier

This study identifies collateral fitness effects of mutations in E. coli, showing that 42% of missense mutations in TEM-1 β-lactamase are deleterious through mechanisms unrelated to protein function.

bioRxiv · Mar 4, 2020Evolutionary Biology