Author

A. L. Young

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Microbiology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:clustering· 1 Research Focus:ecology & biodiversity· 1 Assay:metabolomics· 1 Assay:metagenomics· 1

Frequent Co-Authors

A. Atri· 1 shared A. C. Feeley· 1 shared A. C. Koval· 1 shared A. H. Uribe· 1 shared A. J. Goldenberg· 1 shared A. L. Shergold· 1 shared A. M. L. Crawford· 1 shared B. C. Simmons· 1 shared

Activity by Year

2023-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperPathology

A. Atri

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Disease progression modelling reveals heterogeneity in trajectories of Lewy-type alpha-synuclein pathology

S. E. Mastenbroek, J. W. Vogel, L. E. Collij, G. E. Serrano, C. Tremblay, A. L. Young, R. A. Arce, H. A. Shill, E. D. Driver-Dunckley, S. H. Mehta, C. M. Belden, A. Atri, P. Choudhury, F. Barkhof, C. H. Adler, , O. Hansson

The paper models disease progression in Lewy body disorders using neuropathological data, identifying three distinct trajectories of alpha-synuclein pathology with different clinical associations.

bioRxiv · Dec 5, 2023Pathology

Surveillance of Vermont wildlife in 2021-2022 reveals no detected SARS-CoV-2 viral RNA

H. W. Despres, M. G. Mills, M. M. Schmidt, J. Gov, Y. Perez, M. Jindrich, A. M. L. Crawford, W. T. Kohl, E. Rosenblatt, H. C. Kubinski, B. C. Simmons, M. C. Nippes, A. J. Goldenberg, K. E. Murtha, S. Nicoloro, , Rocka

The study tested 739 Vermont wildlife samples for SARS-CoV-2 and found no viral RNA, contrasting with findings in other North American deer populations.

bioRxiv · Apr 25, 2023Microbiology

Tumour mitochondrial DNA mutations drive aerobic glycolysis to enhance checkpoint blockade

M. Mahmood, E. M. Liu, A. L. Shergold, E. Tolla, J. Tait-Mulder, A. H. Uribe, E. Shokry, A. L. Young, S. Lilla, M. Kim, T. Park, J. L. Manchon, C. Rodriguez-Antona, R. C. Walters, R. J. Springett, , P. A. Gammage

This study shows that mitochondrial DNA mutations in complex I genes drive aerobic glycolysis, enhancing immune checkpoint blockade response in tumors.

bioRxiv · Mar 21, 2023 · PreprintCancer Biology