Author

A. Raina

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 2 Plant Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 2 Organism:Drosophila· 1 Organism:human· 1 Organism:plant (other)· 1

Frequent Co-Authors

A. Majumdar· 1 shared A. Majumdhar· 1 shared H. Joag· 1 shared M. Desai· 1 shared M. Sarkar· 1 shared M. Shinde· 1 shared S. Chinnathambi· 1 shared S. K. Sonawane· 1 shared

Activity by Year

2019-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

A. Majumdar

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

EGCG modulates nuclear formaldehyde-induced Tau phosphorylation in Neuronal cells

S. K. Sonawane, A. Raina, A. Majumdhar, S. Chinnathambi

This study shows EGCG reduces formaldehyde-induced nuclear Tau phosphorylation and CDK5 levels in neuronal cells and a Drosophila model.

bioRxiv · Oct 4, 2020 · PreprintCell Biology

Mutagenic effectiveness and efficiency of gamma rays and sodium azide in M2 generation of Cowpea

A. RAINA, S. Khan

This study evaluates the mutagenic effectiveness and efficiency of gamma rays and sodium azide in cowpea to establish an optimal mutagenesis protocol for genetic improvement.

bioRxiv · Mar 9, 2020 · PreprintPlant Biology

A role of cellular translation regulation associated with toxic Huntingtin protein

H. Joag, V. Ghatpande, M. Sarkar, A. Raina, M. Desai, M. Shinde, T. Bose, A. Majumdar

This study shows that pathogenic Huntingtin protein sequesters the translation regulator Orb2, leading to protein synthesis deficits and lethality, suggesting translation dysfunction as a contributor to Huntington's disease.

bioRxiv · Jul 1, 2019Cell Biology