Author

A. S. Patel

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:drug response / drug discovery· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:clustering· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Fantin· 1 shared A. J. Aguirre· 1 shared A. L. Moriera· 1 shared A. Mondragon· 1 shared A. Sinha· 1 shared A. Smith· 1 shared B. G. Neel· 1 shared B. Moderai· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperPathology

A. Fantin

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

NRP1-expressing myeloid cells promote arteriogenesis in the ischemic limb

J. S. Cho, A. S. Patel, A. Fantin, F. E. Ludwinski, C. Pichette, M. Rudnicki, L. Denti, A. Mondragon, P. Saha, A. Smith, C. Ruhrberg, B. Moderai

The paper identifies NRP1-expressing myeloid cells as promoters of arteriogenesis in ischemic limbs and suggests their therapeutic potential.

bioRxiv · Feb 11, 2024 · PreprintPathology

Adeno-to-squamous transition drives resistance to KRAS inhibition in LKB1 mutant lung cancer

X. Tong, A. S. Patel, E. Kim, H. Li, Y. Chen, S. Li, S. Liu, J. Dilly, K. S. Kapner, Y. Xue, L. Hover, S. Mukhopadhyay, F. Sherman, K. Mynzdar, Y. Gao, , H. Ji

The paper identifies adeno-to-squamous transition (AST) as a mechanism of resistance to KRAS inhibitors in LKB1 mutant lung cancer and links KRT6A expression to poor patient outcomes.

bioRxiv · Sep 7, 2023 · PreprintCancer Biology

Classical oncogene family Myc defines unappreciated distinct lineage states of small cell lung cancer

A. S. Patel, S. Yoo, R. Kong, T. Sato, A. Sinha, L. Bao, M. Fridrikh, K. Emoto, G. Nudelman, C. A. Powell, M. B. Beasley, J. Zhu, H. Watanabe

This study identifies distinct lineage states in small cell lung cancer driven by c-Myc and L-Myc, showing their unique roles in molecular subtypes and histology.

bioRxiv · Nov 3, 2020Cancer Biology