Author

A. Fantin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Pathology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

C. Ruhrberg· 2 shared L. Denti· 2 shared A. Cavalli· 1 shared A. Mondragon· 1 shared A. S. Patel· 1 shared A. Smith· 1 shared B. Balboni· 1 shared B. Moderai· 1 shared

Activity by Year

2022-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersPathology

C. Ruhrberg

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

NRP1-expressing myeloid cells promote arteriogenesis in the ischemic limb

J. S. Cho, A. S. Patel, A. Fantin, F. E. Ludwinski, C. Pichette, M. Rudnicki, L. Denti, A. Mondragon, P. Saha, A. Smith, C. Ruhrberg, B. Moderai

The paper identifies NRP1-expressing myeloid cells as promoters of arteriogenesis in ischemic limbs and suggests their therapeutic potential.

bioRxiv · Feb 11, 2024 · PreprintPathology

Targeting NRP1 with EG00229 induces neurovascular permeability

S. Dragoni, J. T. Brash, A. Fantin, C. Burridge, L. Denti, P. Turowski, C. Ruhrberg

This study shows that EG00229, designed to inhibit VEGF-NRP1 interaction, paradoxically increases vascular leakage by activating NRP1-dependent signaling pathways.

bioRxiv · Nov 7, 2023 · PreprintPhysiology

Metformin antiproliferative activity is exclusively mediated by the membrane functional expression of the Chloride Intracellular Channel 1 in glioblastoma stem cells

I. Verduci, F. Cianci, R. Cazzoli, G. Cannavale, M. Veronesi, B. Balboni, M. Ranucci, L. M. G. Palloni, C. Richichi, S. Faletti, D. Osti, G. Ailuno, G. Caviglioli, C. Tacconi, S. GIROTTO, , M. Mazzanti

This study identifies tmCLIC1 as the specific metformin receptor in glioblastoma stem cells, showing that metformin's antiproliferative effect depends on its binding to tmCLIC1 and subsequent inhibition of mitochondrial respiration.

bioRxiv · Dec 31, 2022 · PreprintCancer Biology