Author

A. Schlientz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Organism:C. elegans· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

A. Bellaart· 1 shared A. C. N. Nguyen· 1 shared A. Desai· 1 shared B. Bowerman· 1 shared C. K. Cahoon· 1 shared D. E. Libuda· 1 shared J. A. Hollis· 1 shared J. Budrewicz· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperCell Biology

A. Bellaart

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2 papers

Recent·Most cited

Cyclin B3 is a dominant fast-acting cyclin that drives rapid early embryonic mitoses

P. Lara-Gonzalez, S. Variyar, J. Budrewicz, A. Schlientz, N. Varshney, A. Bellaart, S. Moghareh, A. C. N. Nguyen, K. Oegema, A. Desai

This study identifies cyclin B3 as the key driver of rapid mitoses in early C. elegans embryos, showing it enables faster cell division compared to cyclin B1 and B2.

bioRxiv · Aug 11, 2023 · PreprintCell Biology

Excess crossovers impede faithful meiotic chromosome segregation in C. elegans

J. A. Hollis, M. L. Glover, A. Schlientz, C. K. Cahoon, B. Bowerman, S. M. Wignall, D. E. Libuda

This study shows that excess crossovers in C. elegans cause chromosome mispatterning and segregation defects, but these errors are corrected by LEM-3 nuclease and homolog tethers during meiosis.

bioRxiv · Jan 30, 2020Cell Biology