Author

A. Shivashankar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

B. S. Choudhary· 2 shared N. Chaudhary· 2 shared N. Verma· 2 shared S. Khanna· 2 shared S. Manna· 2 shared E. Jog· 1 shared J. Barr· 1 shared J. Dani· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersCancer Biology

B. S. Choudhary

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2 papers

Recent·Most cited

EGFR-to-Src family tyrosine kinase switching in proliferating-DTP TNBC cells creates a hyperphosphorylation-dependent vulnerability to EGFR TKI

N. Chaudhary, B. S. Choudhary, A. Shivashankar, S. Manna, K. Ved, S. E. Shaikh, S. Khanna, J. Barr, J. Dani, N. Verma

This study identifies a hyper-phosphorylation state in drug-resistant TNBC cells caused by EGFR-to-Src kinase switching, making them vulnerable to EGFR TKI treatment.

bioRxiv · Jul 17, 2023Cancer Biology

GPX4-VIM equates a proliferating DTP state in TNBC subtypes with converged vulnerabilities to autophagy and glutathione inhibition.

N. Chaudhary, B. S. Choudhary, S. Patra, S. Malvankar, A. Shivashankar, E. Jog, V. K. Kailaje, S. Khanna, S. Manna, S. Sahoo, S. R, M. K. Jolly, S. N. Dalal, N. Verma

The paper identifies a proliferating DTP state in TNBC subtypes that is vulnerable to autophagy and glutathione inhibition, linking GPX4-VIM expression to survival outcomes.

bioRxiv · May 18, 2023 · PreprintCancer Biology