Author

S. N. Dalal

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Bose· 1 shared A. Shivashankar· 1 shared B. S. Choudhary· 1 shared E. Jog· 1 shared K. Modi· 1 shared M. K. Jolly· 1 shared M. Sudarshan· 1 shared N. Chaudhary· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

A. Bose

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2 papers

Recent·Most cited

GPX4-VIM equates a proliferating DTP state in TNBC subtypes with converged vulnerabilities to autophagy and glutathione inhibition.

N. Chaudhary, B. S. Choudhary, S. Patra, S. Malvankar, A. Shivashankar, E. Jog, V. K. Kailaje, S. Khanna, S. Manna, S. Sahoo, S. R, M. K. Jolly, S. N. Dalal, N. Verma

The paper identifies a proliferating DTP state in TNBC subtypes that is vulnerable to autophagy and glutathione inhibition, linking GPX4-VIM expression to survival outcomes.

bioRxiv · May 18, 2023 · PreprintCancer Biology

14-3-3γ prevents centrosome duplication by inhibiting NPM1 function.

A. Bose, K. Modi, S. Dey, S. Dalvi, P. Nadkarni, M. Sudarshan, T. K. Kundu, P. Venkatraman, S. N. Dalal

The paper identifies conserved residues in 14-3-3γ that regulate its interaction with NPM1, affecting centrosome duplication.

bioRxiv · Dec 19, 2019Cell Biology