Author

A. Sukumaran

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Organism:C. elegans· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1

Frequent Co-Authors

J. Geddes-McAlister· 3 shared A. V. Revtovich· 1 shared B. B. Finlay· 1 shared B. Ball· 1 shared B. Muselius· 1 shared B. Raupach· 1 shared C. M. Khursigara· 1 shared E. J. Roach· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersMicrobiology

J. Geddes-McAlister

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Author profile →

3 papers

Recent·Most cited

Integration of homeostatic and adaptive oxidative responses by a putative co-chaperone, Wos2, drives fungal virulence in cryptococcosis

B. Ball, A. Sukumaran, S. Pladwig, S. Kazi, N. Chan, M. Modrakova, J. Geddes-McAlister

This study identifies Wos2 as a co-chaperone that integrates oxidative stress responses in Cryptococcus neoformans, contributing to virulence and offering a potential drug target for antifungal therapy.

bioRxiv · Apr 3, 2023 · PreprintMicrobiology

Dual perspective proteomics infectome profiling discovers Salmonella type III secretion system effector functions in macrophages

J. Geddes-McAlister, A. Sukumaran, S. L. Vogt, J. L. Roland, S. E. Woodward, B. Muselius, L. Gee, E. J. Roach, C. M. Khursigara, B. Raupach, B. B. Finlay, F. Meissner

The paper uses proteomics to identify Salmonella type III secretion system effectors in macrophages and their role in virulence.

bioRxiv · Sep 1, 2021 · PreprintMicrobiology

Systematic characterization and genetic interaction analysis of adhesins in Candida albicans virulence

S. Rosiana, L. Zhang, G. H. Kim, A. V. Revtovich, A. Sukumaran, J. Geddes-McAlister, N. V. Kirienko, R. S. Shapiro

The paper systematically characterizes adhesins in Candida albicans and identifies genetic interactions affecting virulence using a CRISPR-based mutant library and C. elegans model.

bioRxiv · Oct 22, 2020 · PreprintMicrobiology