A. Toleikis

Force generation of KIF1C is impaired by pathogenic mutations

This study investigates the force generation of KIF1C and shows that pathogenic mutations impair its ability to generate force, leading to neuronal dysfunction.

Backstepping mechanism of kinesin-1

This paper proposes that all kinesin-1 backsteps are slips rather than steps, supported by experimental observations of timing differences and load-dependent behavior.

Activation of cytoplasmic dynein through microtubule crossbridging

This study shows that cytoplasmic dynein can crossbridge microtubules, enabling it to slide and polarity-sort microtubule bundles without requiring dynactin or cargo adapters.