Author

A. V. Sarapulov

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

S. Hernandez-Perez· 3 shared M. O. Balci· 2 shared P. Mattila· 2 shared P. Petrov· 2 shared V. Sustar· 2 shared A. Harada· 1 shared B. Tejeda-Gonzalez· 1 shared D. M. Cunha· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersImmunology

S. Hernandez-Perez

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Loss of Rab8a in B cells leads to increased antibody responses and class-switch recombination

S. Hernandez-Perez, A. V. Sarapulov, M. O. Balci, E. Coffey, A. Harada, P. Mattila

This study shows that Rab8a deficiency in B cells increases antibody responses and class-switch recombination by altering signaling pathways.

bioRxiv · Sep 19, 2022 · PreprintImmunology

BCR-induced protein dynamics and the emerging role of SUMOylation revealed by proximity proteomics

L. O. Awoniyi, A. V. Sarapulov, S. Hernandez-Perez, M. Runsala, D. M. Cunha, B. Tejeda-Gonzalez, V. Sustar, M. O. Balci, P. Petrov, P. K. Mattila

This study uses proximity proteomics to identify dynamic protein changes and SUMOylation events in BCR activation, revealing new roles in signaling pathways.

bioRxiv · Sep 29, 2020 · PreprintImmunology

Missing-In-Metastasis / Metastasis Suppressor 1 modulates B cell receptor signaling, B cell metabolic potential and T cell-independent immune responses.

A. V. Sarapulov, P. Petrov, S. Hernandez-Perez, V. Sustar, E. Kuokkanen, R. Vinod, M. Vainio, L. Cords, M. Fritzsche, Y. Carrasco, P. Mattila

This study shows that MIM/MTSS1 regulates B cell receptor signaling, metabolic activity, and T cell-independent immune responses, with implications for cancer biology.

bioRxiv · Mar 8, 2020Immunology